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Spread of oral microorganisms by the circulation

Invasion of the treatment of bacteria and their distribution flow of blood throughout the body is called bacteremia. Bacteremias may arise as a result of surgical and other invasive procedures. They are usually asymptomatic and transient (duration The owner of mononuclear phagocytes and humoral immune response, it is also easy to resolve organisms. Thus, healthy individuals transition bacteremias usually clinical significance and are asymptomatic. However, in individuals who are the lack of proper protection from infection (compromised hosts) bacteria can begin to multiply in the blood as a result of sepsis, a serious disease, which is accompanied by systemic manifestations of inflammation. In compromised hosts (e.g. patients with cancer, uncontrolled diabetes, or immune deficiency), sepsis can start generalized deadly infection.

Acute periapical infections as the origin of metastatic infections

Acute manifestations of endodontic lesions leads to the formation of abscesses in the periapical tissues. Although these lesions often limited to oral region, they can apply to both nearby and distant body compartments along the paths anatomic (fascial plane and space). Therefore, periapical abscess may spread and reach the maxillary sinus, brain, cavernous sinus, eye or mediastinum. Needless to say, some of these conditions are truly dangerous to life. In addition to the direct distribution of pus and bacterial components, brain and lung abscesses may be caused by septic emboli.

Symptomatic apical periodontitis

Symptomatic apical periodontitis may develop as a direct consequence of the decay and infection cellulose within previously healthy periapical region. Then he reflects the answer to the first contact of the periapical tissues periodontal bacteria or use of products derived from infected root canal.

The nature of apical periodontitis

Apical periodontitis performs an important protective function aimed at limiting the bacteria discharged from the root canal space and prevent their spread to adjacent bone marrow spaces and other remote sites. The process is unique in the sense that it cannot eradicate the source of infection. The reason is that once a pulp become necrotic, protective mechanisms may not work far into the root canal due to the absence of cardiovascular support. Although these mechanisms can operate on the apical sidelines of necrotic tissues, they are not able to penetrate the fully developed tooth. Therefore, without proper endodontic treatment apical periodontitis may prevail chronically.

Indications and contraindications for pulp treatment in primary teeth

The most important reason for doing the initial tooth until exfoliation is to preserve the space to prevent overcrowding in permanent dentition. In respect of the molars, as a rule, the most important is the time before the first permanent molars reached occlusion. Other important reasons are the conservation of masticatory function, to prevent a language, habits and preserve the aesthetics.

Glutaraldehyde

Glutaraldehyde (GA) - dialdehyde - gaining increasing attention as a possible replacement for FC as dressing, offer less pulp devitalization but similar clinical results. Glutaraldehyde is not produced commercially, however, the main reason of its instability, even when refrigerated.

Calcium hydroxide

Calcium hydroxide is used as dressing material for both unexposed and exposed to the pulps. This is a strong alkaline mixture with a pH of about 12, causing superficial necrosis about 1.5-2 mm in the area under its placement on the open cellulose. After the initial irritation subcutaneous tissue, pulp produces new collagen and after that bone-as hard tissues. Avoidance extrapulpal blood clot is essential when using calcium hydroxide as dressing, because its presence may hinder pulp healing. It is therefore important to use a gentle equipment, including cutting with high-speed equipment and diamond burs with subsequent irrigation with water or saline in order to achieve hemostasis.

Pulpal inflammation in the primary tooth

Although there has been some controversy in the literature as to the capacity of the pulp milk teeth respond to caries by formation of reparative dentin, several histological studies have shown frequent phenomenon of reparative dentin in primary molars with deep caries(33, 60, 63, 72). Magnusson and Sundell (42) found a significantly lower frequency of cellulose step-by-step exposure with a excavations procedure compared with the direct completion of the excavation deep caries in the primary molars, assuming that the pulp has a good potential for the production of reparative dentin.

Mid-treatment or post-treatment emergency

Painful condition may persist after emergency pulpectomy or you experience the following pulpectomy not initially painful tooth. The last condition is called endodontic exacerbation of the disease. The reason is likely to be bacterial origin, combined with the inadequate technical procedure. Pollution due to the use of rubber dam, unsatisfactory temporary recovery, move carious dentin and plaque in the channel, are key factors(1, 42, 69, 86). Combined with inappropriate intracanal drugs, incomplete instrumentation, non total channels and apical overinstrumentation, easy to understand, that the conditions for bacterial multiplication are created in the system of root canals. It should be emphasized that the complications of this nature should be rare and occur only at a low rate in the correct management of clinical practice (42, 82). Cracked tooth substance and traumatic occlusion and other factors should be considered when examining the patient the reasons for endodontic exacerbation of the disease.

Wound healing after pulpectomy

Healing regularity following pulpectomy characterized by an initial inflammatory reaction in the apical tissue injury caused by the cutting procedure. Residual pulp are often shabby and may even be lost in the process (57). If, by chance, root canal tool was pushed apical hole during a working definition of length or instrument channel, the apical termination of training should still be limited to 1-2 mm from the anatomical apex, to reduce the risk of periapical excess root filling material. In the absence of wound infection, reorganization happens soon. This includes the replacement of the damaged tissue connective tissue derived from the periapical region (39, 59). In the process, some internal or external root resorption may develop, repaired in the future. Patients may experience some tenderness immediately after pulpectomy. These symptoms disappear after a few days, along with the restoration of the apical tissue.
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