There is a wealth of evidence to show the role of dietary sugars in the etiology of dental caries. The evidence comes from many different types of research, including research on humans (both surveillance and intervention), animal studies, human plaque pH studies, enamel plates, and incubation studies of oral bacteria
and food substrates in vitro. Together information from different types of studies, provides an overall picture of the car-iogenic potential of various dietary carbohydrates. The strength of the evidence incriminating sugar in the etiology of dental caries comes from the multiplicity of research, not force any one study alone.
Poor nutrition is only one of many reasons enamel malformations. Enamel defect, which is common in undernourished communities is a linear enamel hypoplasia (LEH). This usually occurs in primary cutters and is characterized by horizontal grooves usually found on the labial surface, which is becoming stained post-eruptively. Several researchers have shown that the presence of enamel hypoplasia is associated with malnutrition and its prevalence increases with the severity of malnutrition. However, the specific mechanism of the influence of diet on the development of enamel hypoplasia was not understood until the 1980s, when the relationship between hypocalcaemia and hypoplasia was discovered. Hypercalcemia is a common in malnutrition and associated with diarrhoea.
In the first half of the twentieth century, it was believed that the provision of good food, and his teeth were developing was the primary way to prevent tooth decay
. It is now known that the local effect of diet in the mouth, teeth after erupted plays a much more important role. However, malnutrition and disadvantages of specific nutrients, affect the development of the teeth and the formation, functions and secretion of salivary glands, which in turn has an influence on the susceptibility to decay.
Mechanical characteristics are not vital dentin: for a long time endodontically treated teeth seemed more fragile due to loss of moisture. Several studies have investigated the mechanical properties of dentin in the vital against the useless teeth. Although moisture content not vary significantly, the compressive and tensile strength not show any significant differences (28). Thus, factors other than dentin features may be more important in the increased fracturing susceptibility end-odontically treat teeth.
In order to achieve long-term clinical success with prosthodontic restoration of the deep filled teeth is important to know the reasons for clinical failure. Some of these reasons, such as recurrent caries or periodontal breakdown, are the same as non-root filled teeth. The main difference is the absence of life cellulose with the capacity to address specific symptoms, which can act as a signal for the patient. To reduce the development of new caries and periodontal disease, individually adapted scheme of preventive care must be installed, including appropriate to recall the schedule.
Due to the possible presence of accessory channels near the floor of the pulp chamber multichannel teeth and the fact that the susceptibility of root canal filling
in the saliva and bacteria seriously deteriorates print quality, use of seals, colored dentin-adhering cement is recommended (8, 22, 42). In case of re-treatment, this material should have a different color of dentin so that the channel can be again, but not to such an extent that it discolors teeth crown.
Microbiota infected necrotic masses tend to dominate the anaerobic bacteria, while facultatives, as a rule, only a small portion of the root canal flora. However, there is a wide variety and large number of individual types and combinations of species may be associated with the development and continuation of apical periodontitis. Therefore, and because the potential for organisms that prevail in biofilms structures on the root canal wall, there is little support for treatment approaches, which selectively focus on specific organisms.
Mechanical devices root canal needs to be supported by frequent watering. There are several important goals, such measures:
- Clean up the garbage and dentinal chips and keep the channels wet, so that the instruments will run smoothly.
- Have antibacterial effect.
- To increase the efficiency of equipment, procedures, by dissolution of the necrotic tissue remains, especially in the areas of mechanical devices cannot reach, including cleft, overlapping channels and accessories.
- Dissolve smear layer.
For optimum results, the RCT, several important steps can be distinguished:
Bacteremia is considered a risk factor for the development of endocarditis. Bacterial endocarditis is a bacterial infection of the heart valves and the epithelial lining (endocardium) of the heart. The term infective endocarditis recently been asked to highlight the fact that microbes than other bacteria can also cause infective endocarditis (25). According to the new terminology, infectious endocarditis), named in honor of an infectious microorganism, for example. streptococcal endocarditis, staphylococcal endocarditis or fungal endocarditis. Although currentlytermed endocardit infection, bacterial endocarditis is still used by many authors in the dental and medical literature.